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. Author manuscript; available in PMC: 2016 Oct 6.
Published in final edited form as: J Immunol. 2015 Aug 14;195(6):2515–2519. doi: 10.4049/jimmunol.1500317

Figure 2. miR-17-92 deficiency reduces the number of virus-specific memory CD4 T cells generated after the primary immune response.

Figure 2

Splenocytes of miR-17-92−/− mice and littermate controls were collected on day 108 p.i. (A) Number of I-AbGP66-77 tetramer+ CD4 T cells and (B) number of IFN-γ+ CD4 T cells per spleen. (C,D) FACS plots of CXCR5 and Ly6C and number of I-AbGP66-77 specific Th1, Ly6C+ TFH, and TFH memory CD4 T cells. (E) Number of I-AbGP66-77 tetramer+ CD4 T cells in liver and lung on day 244 p.i.. Results are representative of at least three independent experiments with n≥3.