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Models for damage avoidance pathways triggered by UV-induced lesions that interfere with DNA replication. a Replication skips the lesion (red triangle), leaving a single-stranded gap that needs filling by homologous recombination. b If the replication fork stalls at a UV-induced lesion, replication fork regression can transfer the lesion back into double-stranded DNA for repair via excision repair. See text for details
