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. 2016 Oct 10;17:790. doi: 10.1186/s12864-016-3143-y

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© The Author(s). 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 3 — a Heat map view of modules clustered based on the module overlap score. b Activation of module clusters (MC1-16) for different phenotypes (P1-2) and chemical classes (C1-6). P1, chemical exposures that cause kidney-cortex, tubule, necrosis; P2, chemical exposures that cause kidney-tubule, regeneration; C1, chemical exposure known to cause hepatotoxicity; C2, fluoroquinolone antibiotics; C3, epithelial growth factor receptor kinase inhibitors; C4, estrogen receptor modulators; C5, high dose of statin drugs; C6, high dose of anti-lipidemic drugs (fibrates)