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. 2016 Oct 14;22(38):8631–8637. doi: 10.3748/wjg.v22.i38.8631

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©The Author(s) 2016. Published by Baishideng Publishing Group Inc. All rights reserved.

This article is an open-access article which was selected by an in-house editor and fully peer-reviewed by external reviewers. It is distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited and the use is non-commercial.

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Pathology of the pancreatic tumor. A: Macroscopic appearance of the resected specimen. The tumor forms a poorly defined, whitish mass lesion measuring 60 mm in diameter. A large cyst filled with hemorrhagic material is present in the central portion of the tumor; B-C: Histology of the tumor. The major part of the tumor is composed of non-cohesive, pleomorphic tumor cells showing a sarcomatous appearance (B), whereas the minor part of the tumor is composed of irregularly shaped fused glandular structures, exhibiting the histology of moderately differentiated adenocarcinoma (C) [hematoxylin and eosin (HE) staining; original magnification, B: × 40, C: × 10]; D: Central part of the tumor. A cystic lesion filled with fluidic material (upper right) is surrounded by necrotic tumor tissue focally containing viable anaplastic carcinoma cells around the blood vessels (lower left/arrow) (HE staining; original magnification, × 4); E, F: Immunohistochemistry. Positive staining for pancytokeratin (AE1/AE3) is observed in both the adenocarcinoma component (E; left) and the sarcomatoid component (E; right), whereas positive vimentin staining is observed in the sarcomatoid component (F; right) and normal stroma but not in the adenocarcinoma component (F; left) (original magnification × 4).