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. 2016 Sep 13;34(3):223–229. doi: 10.3857/roj.2016.01816

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Copyright © 2016 The Korean Society for Radiation Oncology

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/4.0/) which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Fig. 4. — Expression of vascular endothelial growth factor (VEGF) and hypoxia-inducible factor 1 alpha (HIF-1α) decreased in tumor cells when captopril was given concurrently with radiation compared to untreated, irradiated, or captopril-treated tumors. (A) Immunohistochemistry (IHC) staining (brown) of VEGF and HIF-1α in tumor cells of untreated mice (Control), irradiated mice (IR), captopril-treated (Cap) and radiation plus captopril-treated mice (Cap + IR) (H&E, ×10). Relative expression of HIF-1α (B) and VEGF (C) in treated groups compared to control group.