Skip to main content
. 2015 Aug 1;6(4):362–369. doi: 10.1016/j.jtcme.2015.07.002

Fig. 1.

Fig. 1

CAE attenuates Aβ1–40-induced neurotoxicity. Differentiated PC12 (A) and IMR32 (B) cells were treated with varying concentrations of aggregated Aβ1–40, and cell viability was measured at 24 and 48 h after treatment. To determine the effect of CAE on Aβ1–40 neurotoxicity, differentiated PC12 (C) and IMR32 (D) cells were treated with aggregated Aβ1–40 and CAE at indicated concentrations for 24 and 48 h, and cell viability was measured. In all experiments, cells left untreated served as controls. Values are expressed as mean ± SD (n = 6). *Significantly different (P < 0.05) vs. the control group. #Significantly different (P < 0.05) vs. the Aβ1–40 treatment alone.