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. 2016 Jan 12;6(1):e711. doi: 10.1038/tp.2015.191

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Copyright © 2016 Macmillan Publishers Limited

This work is licensed under a Creative Commons Attribution-NonCommercial-NoDerivs 4.0 International License. The images or other third party material in this article are included in the article's Creative Commons license, unless indicated otherwise in the credit line; if the material is not included under the Creative Commons license, users will need to obtain permission from the license holder to reproduce the material. To view a copy of this license, visit http://creativecommons.org/licenses/by-nc-nd/4.0/

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The increase of DNMT1 and TET1 mRNA in the FC of PRS mice is reduced by clozapine but not by haloperidol. Measurements were performed in the same animals tested in Table 1 and Figure 2. The values of mRNA are expressed as % relative to ActB. The data are expressed as mean±s.e.m. of six mice per group. *P<0.05 when vehicle (VEH)-treated PRS samples are compared with PRS clozapine (CLZ)-treated samples or with treated or untreated NS samples. ^#P<0.05 when haloperidol (HAL)-treated PRS samples are compared with PRS clozapine-treated samples or to haloperidol-treated NS samples. One-way ANOVA followed by Student–Newman–Keuls multiple-comparison procedures. ANOVA, analysis of variance; DNMT1, DNA-methyltransferase 1; FC, frontal cortex; mRNA, messenger RNA; NS, nonstressed; PRS, prenatally stressed; TET1, ten-eleven methylcytosine dioxygenase 1.