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. 2016 Oct 19;10:237. doi: 10.3389/fncel.2016.00237

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Copyright © 2016 Wang, Zhou, Bo, Li, Yu, Chen, Zhu and Zhao.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution and reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Effects of CTRP3 treatment on neurological functions 7 days after ICH. All animals after ICH showed significant neurological deficits based on performance on the modified Garcia (A), beam balance (B) and wire hanging (C) tests. Rats treated with rCTRP3 showed reduced neurological deficits in all three tests. Treatment with Lenti-CTRP3 starting 2 weeks before induction of ICH showed a tendency to ameliorate neurological deficits (D–F). Values are mean ± SEM. n = 9 per group. *p < 0.05 vs. sham; **p < 0.01 vs. sham; ^#p < 0.05 vs. vehicle or Lenti.null. CTRP3, C1q/tumor necrosis factor-related protein-3; rCTRP3, recombinant CTRP3; Lenti-CTRP3, Lentivirus overexpression of CTRP3; ICH, intracerebral hemorrhage.