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. 2016 Oct 19;12(10):e1006394. doi: 10.1371/journal.pgen.1006394

Fig 3. Epistasis analysis of RMI2, MUS81 and RTEL1 as determined by cell death and root length analyses.

Fig 3

The relationship of RMI2, MUS81 and RTEL1 in the maintenance of replication-associated repair processes was determined by cell death analysis in root tips and by root length measurement (n = 3). A defect in RMI2 did not lead to any dead TA cells or reduced root length compared to wild-type plants. (A + B) Root lengths in both double mutants rmi2-2 mus81-1 and rmi2-2 rtel1-1 were reduced compared to the corresponding single mutants and wild-type plants. (C + D) The number of dead TA cells adjacent to the stem cell niche in the root meristems was significantly elevated in both double mutants rmi2-2 mus81-1 and rmi2-2 rtel1-1 compared to the corresponding single mutants and wild-type plants (n = 3). Significant differences were calculated using the two-sample t-test, two-sided with no equal variance, p-values: *P ≤ 0.05; **P < 0.01; ***P < 0.001.