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. 2016 Oct 21;13:71. doi: 10.1186/s12986-016-0129-3

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© The Author(s). 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 2 — Serial data from OGTTs. Active glucagon-like-peptide 1 (GLP-1) (a) and gastric inhibitory peptide (GIP) (b) are shown after ingestion of either Diet Rite Cola™ ( with dashed line), Diet Mountain Dew™ ( with dotted line), 68 mg sucralose and 41 mg acesulfame-potassium in seltzer water ( with dashed and dotted line) or seltzer water ( with solid line) 10 min prior to a 75 g oral glucose load. Active GLP-1 was augmented in all three NNS conditions vs. the seltzer water condition. All data are expressed as mean ± standard error

Inline graphic — Serial data from OGTTs. Active glucagon-like-peptide 1 (GLP-1) (a) and gastric inhibitory peptide (GIP) (b) are shown after ingestion of either Diet Rite Cola™ ( with dashed line), Diet Mountain Dew™ ( with dotted line), 68 mg sucralose and 41 mg acesulfame-potassium in seltzer water ( with dashed and dotted line) or seltzer water ( with solid line) 10 min prior to a 75 g oral glucose load. Active GLP-1 was augmented in all three NNS conditions vs. the seltzer water condition. All data are expressed as mean ± standard error