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. 2016 Nov 1;11(11):e0165916. doi: 10.1371/journal.pone.0165916

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© 2016 Eissa et al

This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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Fig 8 — Representative photomicrographs immunohistochemically stained sections of rats’ right hind paws with the anti-foxp3 antibody showing normal control rat ‘s paw in (a) with no inflammatory infiltrate and absent foxp3⁺ cells (x100). Scattered foxp3⁺ cells (↑) with brown stained nuclei of untreated arthritis group are shown in (b) and (c) (x200 and x400, respectively). (d) and (e) show marked increase in the foxp3⁺ cells in joints of ASMA-treated arthritis rat (x200 and x400, respectively). Moderate increase in the number of fox p3⁺ cells after treatment with ATSA is shown in f (x200) with its high power magnification in (g) (x400). ASMA; Autoclaved Schistosoma Mansoni antigen, ATSA; Autoclaved Trichinella Spiralis antigen.