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. 2016 Apr 27;7(24):36510–36528. doi: 10.18632/oncotarget.9058

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Copyright: © 2016 Bai et al.

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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A, B. Increased expression levels of CDK5 and p35 at indicated concentrations of glucose compared with the control (5 mM). C, D. Increased expression levels of CDK5 and p35 at indicated time points in high glucose medium (30 mM) compared with the control (0 h). E. Increased expression levels of CDK5 (red) and p35 (green) at indicated time points. F. Positive correlation between the expression of CDK5 and p35 at indicated time points. G. Increased CDK5 kinase activity in a time-dependent manner compared with the control (0 h). Results are presented as mean ± SD of three independent experiments. *P<0.05, #P<0.001. , CDK5; , p35.

Inline graphic — A, B. Increased expression levels of CDK5 and p35 at indicated concentrations of glucose compared with the control (5 mM). C, D. Increased expression levels of CDK5 and p35 at indicated time points in high glucose medium (30 mM) compared with the control (0 h). E. Increased expression levels of CDK5 (red) and p35 (green) at indicated time points. F. Positive correlation between the expression of CDK5 and p35 at indicated time points. G. Increased CDK5 kinase activity in a time-dependent manner compared with the control (0 h). Results are presented as mean ± SD of three independent experiments. *P<0.05, #P<0.001. , CDK5; , p35.