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. 2016 Oct 25;41(3):287–296. doi: 10.5114/ceji.2016.63129

Table 1.

Characteristics of the study group

Patients N = 86
Sex n %
 Male 55 64
 Female 31 36
Type of HSCT n %
 MUD 59 69
 HLA ID SIB 18 21
 HLA MM REL 9 10
Source of stem cells n %
Peripheral blood 71 83
  – after T cell depletion in vitro 8 9
Bone marrow 15 17
Diagnosis n %
Malignant (M) 64 74
 Acute lymphoblastic leukaemia (ALL) 33 38
 Acute myeloblastic leukaemia (AML) 17 20
 Solid tumours (NBL, WT, PNET) 6 7
 Chronic myeloid leukaemia (CML) 1 1
 Hodgkin lymphoma (HL) 2 2
 Non-Hodgkin lymphoma (NHL) 1 1
 Myelodysplastic syndrome (MDS) 3 3
 Xanthogranuloma juvenile 1 1
Non malignant (NM) 22 26
 Severe aplastic anaemia (SAA) 4 5
 Metabolic diseases (WM) 1 1
 Immune deficiencies (ID*) 17 20
Conditionning
 Myeloablative (MA) 50 58
 Reduced-intensity conditioning (RIC) 24 28
 Non-myeloablative (NMA) 8 10
 None 4 4
Graft versus host disease (GvHD) prophylaxis
 ATG 50 58
 CAMPATH 13 15
 None 22 26
 ATG and CAMPATH 1 1

HLA ID SIB – HLA identical sibling; MUD – matched unrelated donor; HLA MM REL – HLA mismatched related donor; NBL – neuroblastoma; WT – Wilms tumour; PNET – primitive neuroectodermal tumour