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. 2016 Nov 7;35:172. doi: 10.1186/s13046-016-0448-2

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© The Author(s). 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 4 — The role of IL-7R in intracellular pathways, molecules, and EMT in HBX-positive hepatoma cells. a IL-7R shRNA inhibition of IL-7R expression in hepatoma cells stably transfected with HBX. shNC cells, cells transfected with shRNA control vectors; shIL-7R cells, cells transfected with IL-7R shRNA vectors. b The role of IL-7R in the activation of JNK, AKT, and STAT5 in hepatoma cells stably transfected with HBX. HBX-shNC cells, HBX-positive cells transfected with shRNA plasmids. HBX-shIL-7R cells, HBX-positive cells transfected with IL-7R shRNA plasmid. c The function of IL-7R on CyclinD1 and MMP-9 in hepatoma cells stably transfected with HBX. d The role of IL-7R in the EMT of hepatoma cells steadily transfected with HBX