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. 2016 Sep 8;44(19):9358–9368. doi: 10.1093/nar/gkw762

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© The Author(s) 2016. Published by Oxford University Press on behalf of Nucleic Acids Research.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by-nc/4.0/), which permits non-commercial re-use, distribution, and reproduction in any medium, provided the original work is properly cited. For commercial re-use, please contact journals.permissions@oup.com

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Figure 7. — Arginine methylation promotes Scd6-eIF4G1 interaction. (A) Glutathione pull-downs from yeast cells expressing eIF4G1-GST in wild-type and Δhmt1 background. Scd6 was expressed from galactose-inducible plasmid. (B) Quantitation of three independent experiments (n = 3) that were performed as explained in A. (C) Glutathione pull-down from eIF4G1-GST strain expressing wild-type Scd6 or AMD mutant under galactose-inducible promoter. (D) Quantitation of three independent glutathione pull-downs (n = 3) that were performed as described in C.