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. 2016 Oct 5;45(5):1517–1527. doi: 10.1093/ije/dyw218

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© The Author 2016; all rights reserved. Published by Oxford University Press on behalf of the International Epidemiological Association

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/4.0/), which permits non-commercial re-use, distribution, and reproduction in any medium, provided the original work is properly cited. For commercial re-use, please contact journals.permissions@oup.com

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Figure 4. — Network correlation analysis of molecular markers previously tested for association with TCE exposure and metabolic features detected in this study. Only correlations with Spearman |r| ≥ 0.3 and P-value ≤ 0.05 corresponding to identified metabolites or probable halogens were included. Immune markers: CD4 = CD4⁺ T cell count; IgG, immunoglobulin G; IgM, immunoglobulin M; LY, lymphocyte cell count; mtDNA, mitochondrial DNA turnover rate; sCD27, soluble CD27; sCD30, soluble CD30; WBC, white blood cell count. Nephrotoxicity markers: aGST, urinary α glutathione-s-transferase; piGST, urinary pi glutathione-s-transferase; KIM1, urinary kidney injury marker 1; NAG, urinary n-acetyl-beta-glucosaminidase. TCE exposure markers: Conj. TCEOH: urinary trichloroethanol glucuronide; Free TCEOH: urinary trichloroethanol; Tot TCEOH: sum of urinary trichloroethanol and trichloroethanol glucuronide; TCA: urinary trichloroacetic acid.