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. 2016 Oct 12;14(5):4643–4649. doi: 10.3892/mmr.2016.5838

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Copyright: © Yang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 1. — Images captured of the fluorescent staining of Col I and α-SMA in lungs harvested from mice with silica inhalation-induced lung fibrosis treated with emodin and/or nicotinamide. The graph below shows the intensity of Col I and α-SMA fluorescent staining, presented as the mean ± standard deviation. Magnification, ×200. ^aP<0.05, compared with the untreated control; ^bP<0.05, compared with the emodin-treated group; ^cP<0.05, compared with the silica-exposed group; ^dP<0.05, compared with the emodin-treated silica-exposed group. Col I, collagen I; α-SMA, α-smooth muscle actin.