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. 2016 Oct 12;14(5):4643–4649. doi: 10.3892/mmr.2016.5838

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Copyright: © Yang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 4. — Immunoblots of Sirt1, Ac-Smad3, Smad3, Col I and GAPDH in harvested lungs from mice with silica inhalation-induced lung fibrosis treated with emodin and/or nicotinamide. Graphs show the relative expression levels of Sirt1, Ac-Smad3 and Col I. ^aP<0.05, compared with untreated control; ^bP<0.05, compared with emodin-treated group; ^cP<0.05, compared with the silica-exposed group; ^dP<0.05, compared with the emodin-treated silica-exposed group. Sirt1, surtuin 1; Ac-Smad3, acetylated small mothers against decapentaplegic 3; Col, collagen I.