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. 2016 Feb 10;221(9):4615–4629. doi: 10.1007/s00429-016-1192-2

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© The Author(s) 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made.

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Fig. 4 — A. Relative quantification of fos reactivity (mean ± SEM) in the orbitofrontal cortex (OFC), amygdala (Amy), nucleus accumbens (NAcc), basolateral amygdala (BLA), prelimbic (PrL), infralimbic (IL), cingular cortex (Cg), caudate putamen, (CPu), insular cortex (CIns), hippocampus and motor cortex. c-fos quantification was expressed as a percentage of that measured in the control group (dotted line) for safe (n = 5), average (n = 13) and risky (n = 6) mice. Only safe mice differed from the controls for the OFC, NAcc and PrL (W, *p < 0.05). The three groups differed from each other only regarding c-fos activation of the PrL (KW, ^# p < 0.05). b c-fos reactivity was correlated with the percentage of advantageous choices during the 30 last trials (p < 0.05)