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. 2016 Feb 10;221(9):4615–4629. doi: 10.1007/s00429-016-1192-2

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© The Author(s) 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made.

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Fig. 5 — Basal rates of serotonin (5-HT) (a–d) and dopamine (DA) (e–h) in the prelimbic (PrL), the insular cortex (CIns), orbitofrontal cortex (OFC), the hippocampus, the amygdala (Amy), the nucleus accumbens (NAcc) and the caudate putamen (CPu) for safe (n = 16), average (n = 20) and risky (n = 14) mice. Results are expressed as mean ± SEM for each group. *p < 0.05 represented a significant difference between each groups (MW). Safe mice had a low level of 5-HT in the PrL, the CIns and less DA in the Amy and the CPu. Risky mice had a low level of 5-HT in the OFC and a higher level in the hippocampus. Risky mice also had a higher level of DA in the hippocampus. No significant difference existed between groups regarding the NAcc (ns)