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. 2016 Nov 10;16:199. doi: 10.1186/s12886-016-0373-3

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© The Author(s). 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 7 — a Effect of BK on TGF-β1-induced Akt phosphorylation in RPE cells. a TGF-β1 facilitated significant increases in Akt phosphorylation levels in treated RPE cells compared to control cells; however, this effect was inhibited via incubation with BK before TGF-β1 treatment but could be blocked by HOE-140. b Neither BK nor HOE-140 had an effect on TGF-β1-induced Erk1/2 phosphorylation. *P < 0.05 versus without addition, && P < 0.01 versus with TGF-β1 alone. ^#### P < 0.0001 versus with BK and TGF-β1