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. 2016 Nov 11;11(11):e0166184. doi: 10.1371/journal.pone.0166184

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© 2016 Pires-Neto et al

This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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Fig 2 — A: AQP1 in alveolar (short arrow) and arteriolar (long arrow) endothelial cells. Double staining for AQP1 (red) and PII cels (TTF-1, brown, red arrowhead). B: AQP1 in vascular endothelial cells (arrow). C: AQP3 in PII cells (arrows). Double staining for AQP3 (red) and PII cels (TTF1, brown). D: AQP3 in epithelial bronchiolar cells (arrow). E: AQP5 in PI cells lining the alveolar septum (arrow). Double staining for AQP5 (red) and PII cels (TTF1, brown, red arrowhead). F: AQP5 in epithelial bronchiolar cells (long arrow) and submucosal glands (short arrow). G: Na-K-ATPase channel in PI (arrow) and PII (red arrowhead) cells. Double staining for Na-K-ATPase channel (red) and PII cels (TTF1, brown). H: ENaC channel in PI cells lining the alveolar septum (arrow) and PII cells (red arrowhead). I: ENaC channel in epithelial bronchiolar cells (long arrow) and vascular endotelial cells (short arrow).