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. 2015 Dec 11;18(6):913–919. doi: 10.4103/1008-682X.167714

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Copyright: © 2016 AJA, SIMM & SJTU

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-ShareAlike 3.0 License, which allows others to remix, tweak, and build upon the work non-commercially, as long as the author is credited and the new creations are licensed under the identical terms.

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(a) Immunoreactive staining of Ki-67 and apoptotic cancer cells by Tunel in PC3 tumors (b) The ratio of Ki-67-positive cancer cells in PC3 tumors. Fibroblasts induced the expression of Ki-67; finasteride did not further promoted the expression of Ki-67 in the presence of wild fibroblasts. (c) The apoptotic index in PC3 tumors. Fibroblasts induced the PC3 cell apoptosis while finasteride repressed PC3 cell apoptosis in the presence of fibroblasts. c-Jun played a critical role in mediating the apoptosis-repressing effect of finasteride.