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. 2016 Nov 16;7:438. doi: 10.3389/fphar.2016.00438

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Copyright © 2016 Fardoun, Nassif, Issa, Baydoun and Eid.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Genetic basis of RP. The genetic basis of RP is supported by familial studies and twin analysis in addition to a reported case of a 1-month male baby diagnosed with the disease. Furthermore, a combination of positive genotypes for both genes encoding glutathione S-transferase M1 and T1 subtypes may have a role in susceptibility to RP. Linkage analysis pinpointed five areas corresponding to three candidate genes (β-subunit of muscle acetylcholine receptor, 1E and 1B serotonin receptors) which could be associated to RP.