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. 2016 Oct;11(10):1545–1548. doi: 10.4103/1673-5374.193222

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Copyright: © Neural Regeneration Research

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-ShareAlike 3.0 License, which allows others to remix, tweak, and build upon the work non-commercially, as long as the author is credited and the new creations are licensed under the identical terms.

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Schematic representation of the role of oxidative/nitrosative stress & PARP overactivation and therapeutic potential of PARP inhibitors in CCI-induced peripheral neuropathy.

ATP: Adenosine triphosphate; CCI: chronic constriction injury; GSH: glutathione; IL-6: interleukin 6; iNOS: inducible nitric oxide synthase; MDA: malondialdehyde; NAD: nicotinamide adenine dinucleotide; NF-κB: nuclear factor-κB; NO: nitric oxide; PARP: poly (ADP-ribose) polymerase; SOD: superoxidase dismutase; TNF-α: tumor necrosis factor α.