Table 3. Reclassification for 20-year predicted absolute risk of gastric cancer development.
| Number of subjects who developed gastric cancer | ||||
| Basic model with the combination of H. pylori antibody and sPG | ||||
| Basic model with H. pylori antibody | <3.0% | 3.0%–12.0% | >12.0% | Total |
| <3.0% | 8 | 11 | 0 | 19 |
| 3.0%–12.0% | 9 | 25 | 17 | 51 |
| >12.0% | 0 | 8 | 45 | 53 |
| Total | 17 | 44 | 62 | 123 |
| Number of subjects who did not develop gastric cancer | ||||
| Basic model with the combination of H. pylori antibody and sPG | ||||
| Basic model with H. pylori antibody | <3.0% | 3.0%–12.0% | >12.0% | Total |
| <3.0% | 762 | 158 | 3 | 923 |
| 3.0%–12.0% | 298 | 623 | 105 | 1026 |
| >12.0% | 0 | 114 | 200 | 314 |
| Total | 1060 | 895 | 308 | 2263 |
H. pylori, Helicobacter pylori; sPG, serum pepsinogen.
The basic model included age, sex, body mass index, total cholesterol, hemoglobin A1c, smoking habits, and daily total energy and salt intakes.