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. 2016 Jul 8;12(4):611–625. doi: 10.1007/s11302-016-9522-7

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© The Author(s) 2016

Open Access This article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made.

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Fig. 7 — The direct effect of the selective P2X₇R antagonist A438079 on ERG components under different physiological conditions. Values are mean ± SEM percentage of pre-treatment control; n = 10. A438079 (10 μM) was superfused for 20–30 min until the response was stable. a In physiological Krebs medium, application of A438079 alone had no overall effect on the a-wave (i) or b-wave (ii). b In separate experiments using the pharmacologically modified Krebs medium, A438079 induced a marked potentiation in the a-wave (i) compared to control (P < 0.05). The b-wave (ii) was significantly reduced in the presence of A438079 (P < 0.05)