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Potential conjunction of triggers (workload, stress, sadness/anger) and acute risk factors (hemodynamic effects, prothrombotic conditions, inflammatory reactions, and neurohormonal influences) that were ‘superposed’ to disorders already caused by the chronic risk factors and initiate the acute coronary events [13]. BP blood pressure, CNS central nervous system, HR heart rate, PAI-1 plasminogen activator inhibitor-1
