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. 2016 Nov 30;7:471. doi: 10.3389/fphar.2016.00471

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Copyright © 2016 Ding, Li, Guo, Liu, Tong, Pan, Lu, Ye, Chen and Zhang.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Triptolide attenuated the hypertrophic response of neonatal rat ventricular myocytes. Hypertrophic response of neonatal rat ventricular myocytes (NRVM) was induced by angiotensin II (Ang II). (A) NRVM treated with Ang II (1 μmol/L) and triptolide (TP) for 24 h, stained with rhodamine-phalloidin (bar = 50 μm); (B) cell size (n = 50 cells in each group); (C) β-MHC mRNA expression determined using Real-time PCR (n = 4); (D) β-MHC expression level determined using Western blotting (n = 4). The data are presented as mean ± SEM, ^∗∗p < 0.01 compared with the control group; ^#p < 0.05, ^##p < 0.01 compared with the Ang II-treated group (one-way ANOVA).