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. 2016 Dec;75:39–49. doi: 10.1016/j.jaut.2016.07.004

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© 2016 The Authors

This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/).

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Fig. 4 — IDO is required during EAE priming phase (A-C) EAE was induced in WT → WT (■), pIII + IV^−/− → WT (○) and IDO^−/− → WT () BM chimeras. (A) Clinical scores were followed daily (two-way ANOVA with Bonferroni post Hoc test). Frequencies of (B) IFN-γ⁺ (left) and IL-17⁺ (right) CD4⁺ T cells, (C) Ki67⁺ (left), Ki67⁺CD103⁺ICOS⁺ (middle) and IL10⁺ (right) among CD25^hiFoxp3⁺CD4⁺Tregs in dLN at d9 (one-way ANOVA with Bonferroni post Hoc test). (A–C) Results are representative of at least 3 independent experiments with 8 mice per group. Error bars depict mean ± SEM. *P < 0.05, **P < 0.01, ***P < 0.001, NS = Non significant.

Inline graphic — IDO is required during EAE priming phase (A-C) EAE was induced in WT → WT (■), pIII + IV^−/− → WT (○) and IDO^−/− → WT () BM chimeras. (A) Clinical scores were followed daily (two-way ANOVA with Bonferroni post Hoc test). Frequencies of (B) IFN-γ⁺ (left) and IL-17⁺ (right) CD4⁺ T cells, (C) Ki67⁺ (left), Ki67⁺CD103⁺ICOS⁺ (middle) and IL10⁺ (right) among CD25^hiFoxp3⁺CD4⁺Tregs in dLN at d9 (one-way ANOVA with Bonferroni post Hoc test). (A–C) Results are representative of at least 3 independent experiments with 8 mice per group. Error bars depict mean ± SEM. *P < 0.05, **P < 0.01, ***P < 0.001, NS = Non significant.

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