. Author manuscript; available in PMC: 2017 Dec 1.

Published in final edited form as: Cancer Causes Control. 2016 Nov 1;27(12):1419–1428. doi: 10.1007/s10552-016-0820-8

Table 4.

Best fitting models describing the association between life-course body size and risk of type 1endometrial cancer among women not using HT, California Teachers Study, 1995–2012.

Height^a	BMI (age 18)	BMI (baseline)	Weight change	Cases	HR	95% CI
short	normal	normal/ overweight		31	1.0^b
		obese		13	2.39	1.24–4.60

	overweight/obese			11	2.27	1.13–4.54

average	normal	normal	<25 lbs	48	1.04	0.66–1.64
		normal BMI with weight gain ≥25 lbs or overweight		60	1.87	1.21–2.89
		obese		40	2.93	1.82–4.73

	overweight/obese	normal/ overweight		14	1.98	1.05–3.74
		obese		18	3.83	2.11–6.95

tall	normal	normal	<25 lbs	21	1.17	0.67–2.04
		normal BMI with weight gain ≥25 lbs or overweight		34	2.23	1.36–3.64
		obese		21	4.74	2.70–8.32

	overweight/obese			24	4.33	2.51–7.46

Abbreviations: BMI: body mass index; CI: confidence interval; HR: hazard ratio; HT: hormone therapy.

Body size cut-points: height <63 (short), 63–66 (average), ≥67 (tall); BMI (age 18) and BMI <25 (normal), 25–29 (overweight), ≥30 (obese).

Adjusted for the interaction between age at menarche and time-dependent age, gravidity, age at last pregnancy, duration of oral contraceptive use, the interaction between history of hypertension and time-dependent age, history of diabetes, and average daily caloric intake; age was the time metric and the model was stratified by age at baseline.