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. 2016 Apr 8;69(11):1017–1024. doi: 10.1136/jclinpath-2016-203625

Table 1.

Summary of the cohort studied

JAK2V617F and CALR mutation status Total
Subtype JAK2V617F+ CALRMut JAK2V617F−/CALRWT Unknown
Controls 15 15
PV 20 20
ET 26 9 6 31 72
MF* 18 14 5 16 53
Total 64 23 26 47 160

JAK2V617F and CALR lesions did not coexist (ie, JAK2V617F+ and CALRMut were mutually exclusive). All CALR mutations represent frameshift lesions in exon 9 of CALR.

*Includes both primary MF and ET/PV cases having undergone fibrotic transformation.

CALR, calreticulin; ET, essential thrombocythaemia; JAK2, Janus activated kinase 2; MF, myelofibrosis; Mut, mutated; PV, polycythaemia vera; WT, wild type.