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. 2016 Dec 5;2:16070. doi: 10.1038/cddiscovery.2016.70

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Copyright © 2016 The Author(s)

This work is licensed under a Creative Commons Attribution 4.0 International License. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in the credit line; if the material is not included under the Creative Commons license, users will need to obtain permission from the license holder to reproduce the material. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/

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HI brain injury is associated with increased C-subunit levels in the channel-forming fraction. (a) Image of a whole brain stained with TTC (2,3,5-triphenyltetrazolium chloride) from a representative mouse subjected to HI brain injury. Viable tissue is stained red while the infarct is unstained or white. (b) Dot blot for C-subunit prepared using C-subunit antibodies. The extracts from the injured side of the brain showed higher amounts of C-subunit compared with the non-injured side. (c) Densitometry analysis of the C-subunit dot blots of chloroform extracts from brain uninjured (control) and damaged (injury) hemispheres showing increased C-subunit levels in the damaged hemisphere (*P<0.05, Mann–Whitney nonparametric test, n=16 animals).