Table 1.
Patients |
|||
---|---|---|---|
Gene | N | % | |
Activating KIR | 2DS1 | 20 | 38 |
2DS2 | 26 | 49 | |
2DS3* | 14 | 27 | |
2DS4 | 53 | 100 | |
2DS5* | 15 | 29 | |
3DS1 | 18 | 34 | |
Inhibitory KIR | 2DL1 | 51 | 96 |
2DL2 | 26 | 49 | |
2DL3* | 47 | 90 | |
3DL1 | 53 | 100 | |
KIRL* | HLA-C1 | 45 | 87 |
HLA-C2 | 34 | 65 | |
HLA-Bw4 | 41 | 79 | |
2DL1/HLA-C2** | match | 32 | 62 |
mismatch | 18 | 35 | |
2DL2/HLA-C1 | match | 22 | 42 |
mismatch | 3 | 6 | |
2DL3/HLA-C1 | match | 40 | 77 |
mismatch | 7 | 13 | |
3DL1/HLA-Bw4** | match | 41 | 77 |
mismatch | 11 | 21 |
DNA samples of 52 patients were available for analysis.
Due to missing DNA of one patient for KIRL analysis, the total number of KIR/KIRL combinations is reduced by n = 1.