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. Author manuscript; available in PMC: 2016 Dec 9.
Published in final edited form as: J Neurochem. 2012 Apr 27;122(2):321–332. doi: 10.1111/j.1471-4159.2012.07754.x

Figure 5. IVIg treatment increases the level of the anti-apoptotic protein Bcl2 in cultured neurons.

Figure 5

Increasing concentrations of IVIg significantly increased the level of Bcl2 during GD-induced neuronal cell death (a and b). Aβ42 treatment decreased the expression level of Bcl2, however treatment with increasing concentrations of IVIg increased Bcl2 expression level in Aβ42-treated cultures (c and d). **, P < 0.05 compared to vehicle; ***, P < 0.001 compared to vehicle; +, P < 0.05 compared to Aβ42 treated group; +++, P < 0.001 compared to Aβ42-treated group; n = 3 or more biologically different cultures in each group.