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. 2016 Dec 9;9:636. doi: 10.1186/s13071-016-1926-2

Fig. 4.

Fig. 4

Model of triatomine host-microbiota-parasite interactions based on parasite immune modulation. a Triatomine homeostatic responses to blood meal ingestion that control the density of bacteria in the gut. b Immune responses induced by T. cruzi infection control parasite development, and decrease bacteria density; intense competition for nutrients between bacteria and the protozoan occurs in the gut. An overall increase in micobiota diversity concomitant to infection is observed