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. 2016 Dec 13;7:567. doi: 10.3389/fimmu.2016.00567

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Copyright © 2016 Pednekar, Pathan, Paudyal, Tsolaki, Kaur, Abozaid, Kouser, Khan, Peerschke, Shamji, Stenbeck, Ghebrehiwet and Kishore.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Anti-proliferative effects of ghA, ghB, and ghC modules. Human PBMCs from healthy individuals were stimulated with PHA (1 μg/ml) for 3 days with or without recombinant gC1qR (1.25, 2.5, 5, 10, and 20 μg/ml), ghA, ghB, or ghC (20 μg/ml) followed by a 16 h pulse in the presence of tritiated methyl thymidine ([³H]TdR). Each experiment was carried out in triplicates. (A) Each of ghA, ghB, ghC, and gC1qR inhibited PHA-stimulated proliferation of PBMCs; (B) ghA, ghB, and ghC coincubated with gC1qR inhibit PHA-stimulated proliferation of PBMCs.