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. 2016 Dec 15;11(12):e0168505. doi: 10.1371/journal.pone.0168505

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© 2016 Pabbidi et al

This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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Fig 5 — Effects of 4 μM chelerythrine (cheler) on zint-β₂-AR stimulation of I_Ca,L in +PKA(A) and -PKA (B) myocytes and effects of dn-PKCα-Adv (C) on zint-β₂-AR stimulation of I_Ca,L in +LMN myocytes. A; in control +PKA myocytes, chelerythrine had no significant effects on zint-β₂-AR stimulation of I_Ca,L. B; in—PKA myocytes, zint-β₂-AR stimulation elicited a typically enhanced in increase in I_Ca,L compared to controls (A) and chelerythrine significantly inhibited zint-β₂-AR stimulation of I_Ca,L. C; in control +LMN myocytes (βgal) zint-β₂-AR stimulation of I_Ca,L was typically enhanced compared to controls (A). In +LMN myocytes infected with dn-PKCα-Adv, zint-β₂-AR stimulation of I_Ca,L was significantly inhibited. Numbers in parentheses indicate that number of cells studied. * = P<0.05.