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. 2016 Jul 20;44(20):9650–9666. doi: 10.1093/nar/gkw642

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© The Author(s) 2016. Published by Oxford University Press on behalf of Nucleic Acids Research.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by-nc/4.0/), which permits non-commercial re-use, distribution, and reproduction in any medium, provided the original work is properly cited. For commercial re-use, please contact journals.permissions@oup.com

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Figure 5. — Feedback control in OmrA/B expression. (A) Scheme depicting the experiment performed in (B): the β-galactosidase activity of promoter fusions to OmrA, OmrB, ompC and ompF was measured upon overproduction of OmrA, OmrB or of their mut2 derivatives, in strains with a wt copy of ompR (gray bars) or with a mutant version carrying the ompRmut2 compensatory change to OmrA/Bmut2 (black bars). Strains used here are MG1014, MG1666, MG1017, MG1667, MG1901, MG1902, MG1686, MG1688 (panel B, see strain table for details). (C) Activity of the same set of promoter fusions was compared in omrAB⁺ and omrAB⁻ strains in the ompR107 background. Values are represented as % of wt strain for each fusion; the activities of the wt strains were, in Miller units, 135 (P_omrA fusion), 173 (P_omrB), 547 (P_ompF) and 5042 (P_ompC). Strains used are MG1301, MG1908, MG1305, MG1909, MG1696, MG1910, MG1895 and MG1911.