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. 2016 Nov 22;39(1):113–125. doi: 10.3892/ijmm.2016.2809

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Copyright: © Xiong et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Luteolin (Lut) markedly improves pathological inflammatory cell infiltration in the pancreas. Mice were divided into 3 groups (n=18 per group): normal control (NC) group, severe acute pancreatitis (SAP) model group treated with the vehicle (vehicle represents SAP control group) and SAP model group treated with luteolin (Lut). Serum samples and pancreatic tissues were obtained for histological and biochemical analyses at 1, 3 and 6 h after modeling (n=6). (A) Hematoxylin and eosin (H&E) staining, (B) pancreas wet/dry ratios, (C) pathological scores of pancreas tissues, (D) serum amylase levels, (E) serum lipase levels, (F) myeloperoxidase (MPO) levels in pancreas, and (G) malondialdehyde (MDA) levels in pancreas. The results were similar in 3 independent experiments. Data are presented as the means ± SEM. ^aP<0.05 and ^aaP<0.01 vs. NC group; ^bP<0.05 and ^bbP<0.01 vs. vehicle (SAP control group).