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. 2016 Dec 27;7:1964. doi: 10.3389/fpls.2016.01964

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Copyright © 2016 Ouyang, Liu, Huang, Hong, Li, Huang, Zhang, Zhang, Li and Song.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Silencing of SlERF.A3 but not SlERF.A1, SlERF.B4, and SlERF.C3 decreased the resistance against Pst DC3000. pTRV2-SlERF.A1-, pTRV2-SlERF.A3-, pTRV2-SlERF.B4-, pTRV2-SlERF.C3-, or pTRV2-GUS-infiltrated plants were inoculated with Pst DC3000 at 4 weeks after VIGS infiltration. Disease phenotype (A) and bacterial population (B) on leaves of SlERF.A1-, SlERF.A3-, SlERF.B4-, SlERF.C3-, and GUS-silenced plants were recorded and measured. Data presented (B) are the means ± SD from three independent experiments and different letters above the columns indicate significant differences at p < 0.05 level.