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Model for dual modulation of IPCs by the circadian clock and feeding. Inputs from the brain clock can modulate IPC activity both directly via inputs from DN1 neurons and indirectly via clock control of feeding rhythms. Starvation can alter feeding rhythms, leading to altered IPC activity. IPC firing rhythms control insulin-dependent and insulin-independent downstream processes, which include metabolic transcript rhythms in the fat body and behavioral arousal.
