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. 2016 Nov-Dec;49(6):411–412. doi: 10.1590/0100-3984.2015.0139

Figure 1.

Figure 1

A: Axial computed tomography of the chest showing consolidation with air bronchogram and partially rounded hilar opacity, both presenting areas of low signal intensity, in the upper lobe of the left lung. B: Initial MRI of the brain. Scan with T2-weighted turbo spin-echo sequence showing multiple rounded cystic formations of varying dimensions scattered throughout the cerebral and cerebellar parenchyma, as well as in the left thalamus and nucleocapsular regions, having a discrete compressive effect and no signs of significant perilesional edema. C: Follow-up MRI of the brain. Scan with fluid-attenuated inversion recovery sequence, performed five weeks after the start of fluconazole therapy and the restoration of adequate nutrition, showing intense perilesional vasogenic edema indicating a reactive inflammatory process that was nearly undetectable at the beginning of treatment, due to immunosuppression. D: Follow-up MRI of the brain. Scan with fluid-attenuated inversion recovery-weighted sequence, performed five weeks after the start of corticosteroid therapy, when the patient was still under treatment with fluconazole, showing significant regression of the edema around the brain lesions, as well as a reduction in the size of the lesions.