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. 2017 Jan 6;7:39906. doi: 10.1038/srep39906

Table 1. Demographic data and baseline information.

  PDM (n = 61) Control (n = 65) p Value
Group Genotype Pain Interaction
Age, year
 AA homozygotes 22.4 (2.61) 23.2 (1.64) 0.087 0.408 NA NA
 G allele carriers 23.0 (2.04) 24.2 (2.68)
Years of menstruating
 AA homozygotes 11.3 (2.69) 10.5 (1.49) 0.484 0.325 NA NA
 G allele carriers 10.9 (2.60) 12.0 (3.16)
Days of one menstrual cycle
 AA homozygotes 29.4 (1.55) 29.5 (1.25) 0.546 0.760 NA NA
 G allele carriers 29.5 (1.48) 29.5 (1.53)
Edinburgh Handedness
 AA homozygotes 82.5 (17.63) 85.6 (14.35) 0.643 0.291 NA NA
 G allele carriers 82.0 (17.81) 77.3 (20.53)
Mnemonic PRI scores*
 AA homozygotes 34.7 (12.75) NA NA 0.531 0.039 0.300
 G allele carriers 35.0 (14.88) NA
Present PRI scores*
 AA homozygotes 29.5 (12.94) NA
 G allele carriers 33.2 (13.04) NA

Normality was tested for all assessments, and only the PRI scores in the McGill Pain Questionnaire were normality (p > 0.05). The data are presented as the means (SD). NA, not applicable.

*Four subjects with primary dysmenorrhea (PDM; 3 AA homozygotes, 1 G allele carrier) did not complete the pain rating index (PRI) of the McGill Pain Questionnaire and were excluded from these calculations.