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. 2016 Jun 13;7(29):45819–45836. doi: 10.18632/oncotarget.9961

Figure 6. TES functions as a tumor suppressor by activating the p38-MAPK signaling pathway.

Figure 6

A. Representative Western blots showing the phosphorylation and total protein levels of ERK1/2, JNK1/2 and p38 in the genetically modified HCT116 cells. B. Representative Western blots showing the phosphorylation and total protein levels of ERK1/2, JNK1/2 and p38 in the genetically modified DLD-1 cells. GAPDH was used as loading control. ** p < 0.01. Data are plotted as the mean ± SD from five independent experiments. Bars indicate the standard deviation of the mean.