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. 2017 Jan 7;17:35. doi: 10.1186/s12885-016-2998-2

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© The Author(s). 2017

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 2 — Common histologic features of SOS. a Shown are changes typical of sinusoidal obstruction (Case 12). Bridging bands of congestion were observed connecting centrizonal areas (H&E, ×40). b In the same case, a trichrome stain highlights prominent fibrosis extending along centrilobular sinusoids and also shows atrophic hepatocyte trabeculae (trichrome stain, ×200). c Nodular regenerative hyperplasia. In some cases (e.g., Case 6), the hepatic parenchyma showed diffuse transformation into small nodules with little to no fibrosis (reticulin stain, ×40). d Venous occlusion. Case 14 showed complete fibrous obliteration of the central veins (H&E, ×100), which was further confirmed with Verhoeff–Van Gieson staining (inset, ×100)