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. 2017 Jan 6;18:42. doi: 10.1186/s12864-016-3386-7

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© The Author(s). 2017

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 4 — Genotype-common or genotype-specific transcriptional changes in DEGs in the resistant genotype (S18) and the susceptible genotype (P77). a Distribution of genotype-common transcriptional DEGs, modulated in RD and SD datasets into functional categories. b Distribution of genotype-common transcriptional DEGs, modulated in D datasets into functional categories. c Distribution of the genes involved in disease resistance, modulated in both genotypes into functional categories. d The dynamic trends of two DEGs involved in disease resistance. e 22 genotype-specific transcriptional DEGs encoded peroxidase (POD) in S18, 13 genotype-specific transcriptional DEGs encoded peroxidase (POD) in P77. The left side of the red line represents variations in the average fold-changes in genotype-specific transcriptional DEGs in S18, while the right side of the red line represents that in P77