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Aβ42-immunized TgCRND8 mice have a 50% reduction in plaque burden than untreated TgCRND8 mice. Representative pictures of the distribution of Aβ plaques labeled by Dako 6F/3D anti-Aβ antibody in the hippocampus (A and B) and cortex (C and D) of control peptide-immunized (A and C) and Aβ42-immunized TgCRND8 mice (B and D). (Scale bars = 100 μm.)
