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. 2016 Nov 8;12(6):4128–4134. doi: 10.3892/etm.2016.3883

Figure 2.

Figure 2.

ROR2 overexpression inhibited the proliferation of AGS gastric carcinoma cells. The (A) mRNA and (B) protein expression levels of ROR2 in AGS cells transfected with the pcDNA3.1-ROR2 plasmid were examined by reverse transcription-quantitative polymerase chain reaction and western blotting, respectively. (C) MTT assays were performed to determine the proliferation capacity of AGS cells transfected with the pcDNA3.1-ROR2 plasmid. AGS cells transfected with the blank vector and non-transfected AGS cells were used as controls. (D) MTT assays were performed to determine the proliferation capacity of AGS cells transfected with the pcDNA3.1-ROR2 plasmid in the presence or absence of the ROR2 ligand, Wnt5a. **P<0.01, vs. AGS. ROR2, receptor tyrosine kinase-like orphan receptor 2; GAPDH, glyceraldehyde 3-phosphate dehydrogenase; OD, optical density.