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. 2016 Nov 17;3(11-12):322–336. doi: 10.18632/oncoscience.323

Table 2. The loss of Brg1 and/or Brm can enhance cancer development via the up-regulation (as measured by qPCR) of 26 potential cancer-promoting genes from at least 5 categories (angiogenesis, anti-apoptosis, cancer progression, metastasis and proliferation).

Gene Name Aliases Fold Change (Up) Gene Function Other Functions
Brm null Brg1 KO Doubles (DKO)
Itga2 CD49B, DX5 1.4 3.3 4.3 Angiogenesis Metastasis
Sema7A Sema-L; semaphorin 2.3 3.5 4.2 Angiogenesis Metastasis
Serpine1 PAI-1 3.4 5.1 8.9 Angiogenesis Anti-apoptosis
Ear2 Rnase2, Raf3 1.3 8.7 9.6 Anti-apoptosis
Hecw1 Nedl1 2.2 4.4 5.1 Anti-apoptosis
Xaf1 Fbox39 4.0 1.7 5.9 Anti-apoptosis
Ddx46 PRPF5 9.5 3.5 10.2 Cancer progression
Epcam CD326, EGP,
gp40
2.1 4.0 5.2 Cancer progression
Macc Gm267 1.7 3.3 5.7 Cancer progression
Tmprss6 IRIDA 1.8 7.3 9.7 Cancer progression
Dkk1 mdkk-1 1.1 5.4 7.4 Metastasis
Itga3 CD49C, GAPB3 4.4 3.1 7.1 Metastasis
Pdpn GP38, RANDAM-2 Metastasis
Plau u-PA 9.3 2.0 8.8 Metastasis Tamoxifen-Sensitive
SerpinB2 PAI-2,
ovalbumin
1.0 6.1 6.3 Metastasis
SerpinB5 PI-5, Maspin 1.1 3.2 3.5 Metastasis
Serpine1 PAI-1 2.0 3.2 4.8 Metastasis
Etv1 ER81,
Etsrp81
4.9 1.5 6.0 Proliferation Oncogene
Hapln1 CLP, LP-1 0.7 1.8 5.3 Proliferation Oncogene
MycN Nmyc-1 4.4 2.3 6.5 Proliferation Oncogene
Areg Mcub, Sdgf 4.1 1.6 4.5 Proliferation Oncogene
Grem1 Drm 3.0 1.7 4.6 Proliferation Oncogene
Ros1 c-ros 2.6 2.8 5.3 Proliferation Oncogene
Mcoln3 TRPML3 2.2 2.3 4.4 Proliferation
Maf -- 2.0 6.0 8.1 Proliferation Oncogene
Muc1 CD227, EMA 1.4 3.0 3.9 Proliferation Metastasis

The gene name and alias (when applicable) are given in columns 1 and 2, respectively. However, the fold induction of gene expression that was observed in BRM-null, BRG1-KO or DKO-derived tumors compared with wild type tumors is given in columns 3, 4 and 5, respectively. The potential gene function and/or other potential gene functions are listed in columns 6 and 7, respectively.